Seismic Images Show Dinosaur-killing Meteor Made Bigger Splash

The most detailed three-dimensional seismic images yet of the Chicxulub crater, a mostly submerged and buried impact crater on the Mexico coast, may modify a theory explaining the extinction of 70 percent of life on Earth 65 million years ago.

The Chicxulub crater was formed when an asteroid struck on the coast of the Yucatan Peninsula. Most scientists agree the impact played a major role in the "KT Extinction Event" that caused the extinction of most life on Earth, including the dinosaurs.

According to Sean Gulick, a research scientist at the Institute for Geophysics at The University of Texas at Austin's Jackson School of Geosciences and principal investigator for the project, the new images reveal the asteroid landed in deeper water than previously assumed and therefore released about 6.5 times more water vapor into the atmosphere.

The impact site also contained sulfur-rich sediments called evaporites, which would have reacted with water vapor to produce sulfate aerosols. According to Gulick, an increase in the atmospheric concentration of the compounds could have made the impact deadlier in two ways: by altering climate (sulfate aerosols in the upper atmosphere can have a cooling effect) and by generating acid rain (water vapor can help to flush the lower atmosphere of sulfate aerosols, causing acid rain). Earlier studies had suggested both effects might result from the impact, but to a lesser degree.

"The greater amount of water vapor and consequent potential increase in sulfate aerosols needs to be taken into account for models of extinction mechanisms," says Gulick.

An increase in acid rain might help explain why reef and surface dwelling ocean creatures were affected along with large vertebrates on land and in the sea. As it fell on the water, acid rain could have turned the oceans more acidic. There is some evidence that marine organisms more resistant to a range of pH survived while those more sensitive did not.

Gulick says the mass extinction event was probably not caused by just one mechanism, but rather a combination of environmental changes acting on different time scales, in different locations. For example, many large land animals might have been baked to death within hours or days of the impact as ejected material fell from the sky, heating the atmosphere and setting off firestorms. More gradual changes in climate and acidity might have had a larger impact in the oceans.

Gulick and collaborators originally set out to learn more about the trajectory of the asteroid. They had hoped the crater's structure in the subsurface would hold a tell-tale signature. Instead, the structure seemed to be most strongly shaped by the pre-impact conditions of the target site.

"We discovered that the shallow structure of the crater was determined much more by what the impact site was like before impact than by the trajectory of the impactor," says Gulick.

If scientists can determine the trajectory, it will tell them where to look for the biggest environmental consequences of impact, because most of the hazardous, shock-heated and fast-moving material would have been thrown out of the crater downrange from the impact.

Researchers at Imperial College in London are already using computer models to search for possible signatures in impact craters that could indicate trajectory regardless of the initial surface conditions at the impact site.

"As someone who simulates impact events using computers, this work provides valuable new constraints on both the pre-impact target structure and the final geometry of the cratered crust at Chicxulub," says Gareth Collins, a research fellow at Imperial College.

The study "Importance of pre-impact crustal structure for the asymmetry of the Chicxulub impact crater" appears in the February 2008 print edition of the journal Nature Geoscience.

Collaborators on the project included Gail Christeson of the Institute for Geophysics, Penny Barton at the University of Cambridge, Joanna Morgan and Mike Warner at Imperial College, and several graduate students.

Cell Phone Sensors Detect Radiation To Thwart Nuclear Terrorism

Researchers at Purdue University are working with the state of Indiana to develop a system that would use a network of cell phones to detect and track radiation to help prevent terrorist attacks with radiological "dirty bombs" and nuclear weapons.

Such a system could blanket the nation with millions of cell phones equipped with radiation sensors able to detect even light residues of radioactive material. Because cell phones already contain global positioning locators, the network of phones would serve as a tracking system, said physics professor Ephraim Fischbach. Fischbach is working with Jere Jenkins, director of Purdue's radiation laboratories within the School of Nuclear Engineering.

"It's the ubiquitous nature of cell phones and other portable electronic devices that give this system its power," Fischbach said. "It's meant to be small, cheap and eventually built into laptops, personal digital assistants and cell phones."

The system was developed by Andrew Longman, a consulting instrumentation scientist. Longman developed the software for the system and then worked with Purdue researchers to integrate the software with radiation detectors and cell phones. Cellular data air time was provided by AT&T.

The research has been funded by the Indiana Department of Transportation through the Joint Transportation Research Program and School of Civil Engineering at Purdue.

"The likely targets of a potential terrorist attack would be big cities with concentrated populations, and a system like this would make it very difficult for someone to go undetected with a radiological dirty bomb in such an area," said Longman, who also is Purdue alumnus. "The more people are walking around with cell phones and PDAs, the easier it would be to detect and catch the perpetrator. We are asking the public to push for this."

Tiny solid-state radiation sensors are commercially available. The detection system would require additional circuitry and would not add significant bulk to portable electronic products, Fischbach said.

The technology is unlike any other system, particularly because the software can work with a variety of sensor types, he said.

"Cell phones today also function as Internet computers that can report their locations and data to their towers in real time," Fischbach said. "So this system would use the same process to send an extra signal to a home station. The software can uncover information from this data and evaluate the levels of radiation."

The researchers tested the system in November, demonstrating that it is capable of detecting a weak radiation source 15 feet from the sensors.

"We set up a test source on campus, and people randomly walked around carrying these detectors," Jenkins said. "The test was extremely safe because we used a very weak, sealed radiation source, and we went through all of the necessary approval processes required for radiological safety. This was a source much weaker than you would see with a radiological dirty bomb."

Officials from the Indiana Department of Transportation participated in the test.

"The threat from a radiological dirty bomb is significant, especially in metropolitan areas that have dense populations," said Barry Partridge, director of INDOT's Division of Research and Development.

Long before the sensors would detect significant radiation, the system would send data to a receiving center.

"The sensors don't really perform the detection task individually," Fischbach said. "The collective action of the sensors, combined with the software analysis, detects the source. The system would transmit signals to a data center, and the data center would transmit information to authorities without alerting the person carrying the phone. Say a car is transporting radioactive material for a bomb, and that car is driving down Meridian Street in Indianapolis or Fifth Avenue in New York. As the car passes people, their cell phones individually would send signals to a command center, allowing authorities to track the source."

The signal grows weaker with increasing distance from the source, and the software is able to use the data from many cell phones to pinpoint the location of the radiation source.

"So the system would know that you were getting closer or farther from something hot," Jenkins said. "If I had handled radioactive material and you were sitting near me at a restaurant, this system would be sensitive enough to detect the residue. "

The Purdue Research Foundation owns patents associated with the technology licensed through the Office of Technology Commercialization.

In addition to detecting radiological dirty bombs designed to scatter hazardous radioactive materials over an area, the system also could be used to detect nuclear weapons, which create a nuclear chain reaction that causes a powerful explosion. The system also could be used to detect spills of radioactive materials.

"It's impossible to completely shield a weapon's radioactive material without making the device too heavy to transport," Jenkins said.

The system could be trained to ignore known radiation sources, such as hospitals, and radiation from certain common items, such as bananas, which contain a radioactive isotope of potassium.

"The radiological dirty bomb or a suitcase nuclear weapon is going to give off higher levels of radiation than those background sources," Fischbach said. "The system would be sensitive enough to detect these tiny levels of radiation, but it would be smart enough to discern which sources posed potential threats and which are harmless."

The team is working with Karen White, senior technology manager at the Purdue Research Foundation, to commercialize the system. For more information on licensing the cell phone sensor technology, contact White at (765) 494-2609

Protein Discovered That Prevents HIV From Spreading

In a study that could open up the field of virology to an entirely new suite of possibilities and that paves the way for future drug research, scientists at Rockefeller University and the Aaron Diamond AIDS Research Center have pinned down a molecule on the surface of human cells that helps keep particles of mutant strains of HIV from spreading. Rather than floating off to infect more cells, the protein contains the virus particles by keeping them attached to the parent cell’s outer membrane, as if stuck there with glue.

Two years ago, Paul Bieniasz — head of the Laboratory of Retrovirology and ADARC scientist — discovered that normal HIV-1 particles are able to extricate themselves from the sticky membrane surface using a protein called Vpu. Bieniasz has been searching for the source of the glue itself ever since. Now, in an advanced online publication in Nature, he and his colleagues report that they found it: a protein they dubbed “tetherin” for its ability to keep viruses tied to a cell.

“All we knew when we started this two and a half years ago was that a virus lacking Vpu was released less efficiently from cells.” Bieniasz says. “And we had some electron micrographs that showed virus particles stuck there on the surface and clustered inside cells.” Once they started looking carefully at the reasons behind this, they found an antiviral mechanism keeping the HIV-1 mutant particles tethered to the cell. And it wasn’t just HIV — the glue appeared to interfere with the spread of other membrane-encapsulated (or “enveloped”) viruses, too.

To track down the cause of stickiness — and the likely reason HIV evolved Vpu — Bieniasz and his team looked at gene activity across all known human genes, making comparisons between cells that require Vpu for HIV-1 release and those that don’t. Ultimately, they narrowed it down to one very likely candidate. And the candidate, the tetherin protein, passed all the tests the researchers threw at it: When Vpu was not present but tetherin was, large numbers of virus particles piled up on the cell surface. When tetherin was missing, however, even the Vpu-deficient viruses were able to escape.

“We’ve discovered a new way that cells defend themselves against viruses,” Bieniasz says. “I think this will open up a new area of study in virology: how this protein antagonizes other viruses, and how viruses learn to get around it.” Going forward, his lab will focus on how broad tetherin’s antiviral activity is, and whether variations of it exist that might confer additional immunity or sensitivity to HIV and other viruses. And, he notes, if drug researchers are able to interfere with the interaction between tetherin and Vpu, their newly discovered protein might even provide a potential therapeutic target.